When Einstein was about age 3, his parents brought him to a pediatrician because he was not yet talking. Researchers have learned that Einstein had developmental dyslexia. More than a century ago, it was found that lesions of the left angular gyrus—ie, Brodmann’s area 39—induce acquired alexia. Therefore, it is possible that people with developmental dyslexia may also have abnormalities in this region, Kenneth M. Heilman, MD, suggested in his lecture at the 17th Annual Meeting of the American Neuropsychiatric Association. In his view, however, the high ratio of glial cells to neurons that was reported by Diamond et al was less a sign of Einstein’s dyslexia than an indication of the high degree of what Dr. Heilman refers to as “connectivity.”
After viewing photographs taken of Einstein’s brain before its dissection in 1955, Witelson and colleagues noted that Einstein had an enlarged left inferior and—unlike most human beings—undivided parietal lobe, suggesting that this bigger and more highly connected supramodal cortex gave Einstein an advantage in doing mathematics and spatial computations. In 1985, Geschwind and Galaburda posited that delay in the development of the left hemisphere of the brain may allow the right hemisphere, which mediates spatial computations, to become highly specialized. It was Einstein’s view that his own creativity was heavily dependent on spatial reasoning. Thus, the abnormal development of his left hemisphere may have led to the right hemisphere becoming highly specialized for spatial computations, Dr. Heilman theorized.
Read more: http://www.neuropsychiatryreviews.com/may06/einstein.html
